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Histidine Focused Covalent Library

For a long time, irreversible covalent inhibitors have been in the shadows of medicinal chemistry and drug discovery efforts due to their potential toxicity and related safety concerns. However, the approval of 8 covalent drugs over the past decade has restarted the active investigation for new targeted covalent inhibitors (TCIs). One of the main directions of the modern covalent inhibition research in drug development is focused on less studied amino acids (e.g. His, Arg, Met) and their potential to react with the new generation of TCIs irreversibly or reversibly [1].

Side-selective modification of histidine residue remains one of the most difficult and interesting challenges in covalent drug discovery due to its unique imidazole side chain. The nucleophilic heteroaromatic ring is mainly modified by the attack of the electrophiles and different N-substitutional reactions. Nonetheless, this approach remains vulnerable due to the possible covalent binding to serine and cysteine amino acid residues instead [2]. Therefore, the variety of chemoselective covalent modifiers for the histidine amino acid moiety is limited, covering the following structural moieties as potential covalent warheads: 2-cyclohexenone derivatives, and alkyl halides (chlorine derivatives are preferred), sulfonyl fluorides, α-сyanoenones, epoxides, and promising spiro-epoxides [3-7].

The Life Chemicals proprietary Histidine-focused Screening Compound Library comprises over 5,600 covalently binding molecules containing specific structure moieties that could react reversibly or irreversibly (by forming covalent bonds) with histidine residues of a drug target for efficient covalent screening. These small-molecule screening compounds are potential histidine-specific covalent inhibitors, selected from the Life Chemicals HTS Compound Collection based on the 13 most interesting covalent warheads that were reported in several articles [3-10]. They are split, herein, into a number of classes for convenient and quick search:

  • α,β-unsaturated ketones [13]
  • α,β-unsaturated sulfones [11-12]
  • α-cyanoacrylamides [11-12]
  • α-сyanoenones [14]
  • 2-cyclohexenone derivatives [15]
  • acetylenes [16]
  • acrylonitriles [17]
  • aldehydes [18]
  • alkyl halides [19]
  • chloromethyl sulfones [11-12]
  • epoxides/spiro-epoxides [20]
  • sulfonate esters [21]
  • sulfonyl fluorides [22]

The compound selection can be customized based on your requirements, cherry picking is available.

Please, contact us at orders@lifechemicals.com for any additional information and price quotations.

Further exploring our related products will make your search even more rewarding:
 

 Covalent warheads distribution for compounds in the Histidine-focused Covalent Inhibitor Library

Figure 1. Covalent warheads distribution for compounds in the Histidine-focused Covalent Inhibitor Library

Representative compounds from the Histidine-focused Covalent Inhibitor Library

References

  1. Sutanto F., Konstantinidou M., Dömling A. (2020) Covalent inhibitors: a rational approach to drug discovery. RSC Med Chem.;11(8):876-884. DOI: 10.1039/d0md00154f.
  2. Brosnan M., Brosnan J. (2020) Histidine Metabolism and Function, The Journal of Nutrition, Volume 150, Issue Supplement_1, P 2570S-2575S, https://doi.org/10.1093/jn/nxaa079.
  3. Joshi, P. N., & Rai, V. (2018). Single-site labeling of histidine in proteins, on-demand reversibility, and traceless metal-free protein purification. Chemical Communications. doi:10.1039/c8cc08733d.
  4. Sornay, Ch., Vaur, V., Wagner, A. (2022) An overview of chemo- and site-selectivity aspects in the chemical conjugation of proteins. Soc. open sci. 9211563211563. http://doi.org/10.1098/rsos.211563.
  5. Jia, S., He, D., & Chang, C. J. (2019). Bioinspired Thiophosphorodichloridate Reagents for Chemoselective Histidine Bioconjugation. Journal of the American Chemical Society. doi:10.1021/jacs.8b11912.
  6. Narayanan, A., & Jones, L. H. (2015). Sulfonyl fluorides as privileged warheads in chemical biology. Chemical science, 6(5), 2650–2659. https://doi.org/10.1039/c5sc00408j.
  7. Gehringer, M., & Laufer, S. A. (2018). Emerging and Re-Emerging Warheads for Targeted Covalent Inhibitors: Applications in Medicinal Chemistry and Chemical Biology. Journal of Medicinal Chemistry. doi:10.1021/acs.jmedchem.8b01153.
  8. Adusumalli, S. R., Rawale, D. G., Singh, U. (2018). Single-site labeling of native proteins enabled by a chemoselective and site-selective chemical technology. Journal of the American Chemical Society. doi:10.1021/jacs.8b10490.
  9. Powers, J. C., Asgian, J. L., Ekici, Ö. D., & James, K. E. (2002). Irreversible Inhibitors of Serine, Cysteine, and Threonine Proteases. Chemical Reviews, 102(12), 4639–4750. doi:10.1021/cr010182v.
  10. Gehringer, M., & Laufer, S. A. (2018). Emerging and Re-Emerging Warheads for Targeted Covalent Inhibitors: Applications in Medicinal Chemistry and Chemical Biology. Journal of Medicinal Chemistry. doi:10.1021/acs.jmedchem.8b01153.
  11. Ray, S., & Murkin, A. S. (2019). New Electrophiles and Strategies for Mechanism-Based and Targeted Covalent Inhibitor Design. Biochemistry. doi:10.1021/acs.biochem.9b00293.
  12. Benton, P. M. C., Mayer, S. M., Shao, J. (2001). Interaction of Acetylene and Cyanide with the Resting State of Nitrogenase α-96-Substituted MoFe Proteins. Biochemistry, 40(46), 13816–13825. doi:10.1021/bi011571m.
  13. Lonsdale R, Reetz MT. Reduction of α,β-Unsaturated Ketones by Old Yellow Enzymes: Mechanistic Insights from Quantum Mechanics/Molecular Mechanics Calculations. J Am Chem Soc. 2015;137(46):14733-14742. doi:10.1021/jacs.5b08687
  14. Gehringer, M., & Laufer, S. A. (2018). Emerging and Re-Emerging Warheads for Targeted Covalent Inhibitors: Applications in Medicinal Chemistry and Chemical Biology. Journal of Medicinal Chemistry. doi:10.1021/acs.jmedchem.8b01153
  15. Rudolph K, Bauer P, Schmid B, Mueller-Uri F, Kreis W. Truncation of N-terminal regions of Digitalis lanata progesterone 5β-reductase alters catalytic efficiency and substrate preference. Biochimie. 2014;101:31-38. doi:10.1016/j.biochi.2013.12.010
  16. Lee CJ, Liang X, Chen X, et al. Species-specific and inhibitor-dependent conformations of LpxC: implications for antibiotic design. Chem Biol. 2011;18(1):38-47. doi:10.1016/j.chembiol.2010.11.011
  17. Paul AS, Islam R, Parves MR, et al. Cysteine focused covalent inhibitors against the main protease of SARS-CoV-2. J Biomol Struct Dyn. 2022;40(4):1639-1658. doi:10.1080/07391102.2020.1831610
  18. Ma Y, Yang KS, Geng ZZ, et al. A multi-pronged evaluation of aldehyde-based tripeptidyl main protease inhibitors as SARS-CoV-2 antivirals. Eur J Med Chem. 2022;240:114570. doi:10.1016/j.ejmech.2022.114570
  19. Teixeira KN, Oliveira JS, Drabowski B, Bruña-Romero O, Santos AM, Santoro MM. Analysis of the oxidase activity induced by CCl(4) and H(2)O(2) in different recombinant myoglobins. Int J Biol Macromol. 2010;47(2):276-282. doi:10.1016/j.ijbiomac.2010.04.008
  20. Zurita J, Motwani HV, Ilag LL, et al. Detection of Benzo[a]pyrene Diol Epoxide Adducts to Histidine and Lysine in Serum Albumin In Vivo by High-Resolution-Tandem Mass Spectrometry. Toxics. 2022;10(1):27. Published 2022 Jan 8. doi:10.3390/toxics10010027
  21. Delort E, Nguyen-Trung NQ, Darbre T, Reymond JL. Synthesis and activity of histidine-containing catalytic peptide dendrimers. J Org Chem. 2006;71(12):4468-4480. doi:10.1021/jo060273y
  22. Narayanan A, Jones LH. Sulfonyl fluorides as privileged warheads in chemical biology. Chem Sci. 2015;6(5):2650-2659. doi:10.1039/c5sc00408j
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